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J. Clin. Invest. 98(12), 2894-2902. Particle-mediated gene transfer with Transforming Growth Factor β1 cDNAs enhances wound repair in rat skin. 1996

Benn, S., Whitsitt, J., Broadley, K., Nanney, L., Perkins, D., Patel, L., Morgan, J., Swain, W., Davidson, J.

Notes: pGL2 Vector, Beetle Luciferin, Wizard® Plus Maxipreps DNA Purification System, RNasin® Ribonuclease Inhibitor, RQ1 RNase-Free DNase, AMV Reverse Transcriptase and Taq DNA Polymerase were used in this paper. (1963)

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J. Clin. Invest. 98(8), 1851-1859. Prevention of adoptively transferred diabetes in nonobese diabetic mice with IL-10-transduced islet-specific Th1 lymphocytes: A gene therapy model for autoimmune diabetes. 1996

Moritani, M., Yoshimoto, K., Ii, S., Kondo, M., Iwahana, H., Yamaoka, T., Sano, T., Nakano, N., Kikutani, H. and Itakura, M.

Notes: Promega's RQ1 RNase-Free DNase, M-MLV Reverse Transcriptase and RNasin® Ribonuclease Inhibitor were used in this study. (2020)

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FEBS Lett. 343(1), 11-14. Modulation of cytosolic RNase activity by endogenous RNase inhibitor in rat vaginal epithelial cells on estradiol administration. 1994

Rao, K.S., Sirdeshmukh, R. and Gupta, P.D.

Notes: The cytosolic, alkaline RNase in rat vaginal epithelial cells (VEC) from normal, immature rats was found to be present largely in free, active form unlike in many other mammalian tissues where it is known to be present in a latent form as a complex with RNase inhibitor (RNasin). Estradiol (E2) administration induced expression of RNasin activity in the VEC from such animals and caused virtually total inhibition of cytosolic RNase activity in these cells by 12 h after hormone injection. These changes may have metabolic implications in relation to other biochemical events stimulated by estradiol in rat VEC. (1647)

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PCR Methods Appl. 3(6), 317-319. Rapid RT-PCR amplification from limited cell numbers. 1994

Edmands, S., Kirk, J., Lee, A., and Radich, J.

Notes: The inhibitor was used for protection of cells and colonies following RT-PCR. (1672)

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FEBS Lett. 328(3), 250-252. Endotoxin induces rat lung ribonuclease activity. 1993

Clerch, L.B., Wright, A. and Massaro, D.

Notes: Lipopolysaccharide (endotoxin), a component of gram-negative bacteria, causes marked alterations in eukaryotic gene expression and cellular physiology. We show that within one hour of injection of endotoxin into adult rats, there is an induction of ribonuclease activity in the lung. The degradation of RNA was prevented by treatment of the lung extract from endotoxin-injected rats with ribonuclease inhibitor (RNasin). We suggest that induction by endotoxin of ribonuclease activity is a novel mechanism by which cells could alter gene expression to meet an environmental challenge and caution that the presence of ribonuclease can hinder molecular biological analyses of tissue extracts from endotoxin-treated rats. (1644)

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Genes Dev. 6, 1740–51.

Concentration-dependent activities of the even-skipped protein in Drosophila embryos.


Manoukian, A.S., Krause, H.M,

Notes: RNasin Ribonuclease Inhibitor was used in the mRNA and protein localization steps. (4183)

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J. Biol. Chem. 267, 21982-21986. Angiogenin is a cytotoxic, tRNA-specific ribonuclease in the RNase A superfamily. 1992

Saxena, S.K., Rybak, S.M., Davey, R.T., Jr, Youle, R.J., and Ackerman, E.J.

Notes: Protein synthesis was restored to angiogenin-injected oocytes by injecting RNasin® Ribonuclease Inhibitor plus total Xenopus or calf liver tRNAs. (1635)

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J. Biol. Chem. 266(31), 21208-21214. Comparison of RNases and toxins upon injection into Xenopus oocytes. 1991

Saxena, S.K., Rybak, S.M., Winkler, G., Meade, H.M., McGray, P., Youle, R.J. and Ackerman, E.J.

Notes: Recombinant angiogenin injected into oocytes abolished protein synthesis, and this toxic effect was inhibited by RNasin® Ribonuclease Inhibitor. (2248)

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